Mechanism of change in cardiac function after mitral valve replacement

钱伟; 张宝仁; 朱家麟; 陈如坤; 郝家骅; 蔡凯华; 刘振华

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Mechanism of change in cardiac function after mitral valve replacement

Updated：2026-03-12

- Mechanism of change in cardiac function after mitral valve replacement
- Mechanism of change in cardiac function after mitral valve replacement
- 解放军医学杂志（英文版） 1992年第3期页码：256-260
- Affiliations：
  
  1. Departments of Thoracic and Cardiovascular Surgery Changhai Hospital
  2. Second Military Medical University
  3. ,Shanghai,200433
  4. Departments of Thoracic and Cardiovascular and Pathology
  5. Department of Pathology
- Author bio：
- Funds：
  
  Supported by the Youth Foundation of the General Logistics Department of PLA
- DOI：
  中图分类号：
- 纸质出版：1992
- Accepted：
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Mechanism of change in cardiac function after mitral valve replacement[J]. 解放军医学杂志（英文版）, 1992,(3):256-260.

[1]钱伟,张宝仁,朱家麟,陈如坤,郝家骅,蔡凯华,刘振华.Mechanism of change in cardiac function after mitral valve replacement[J].Journal of Medical Colleges of PLA,1992(03):256-260.
Mechanism of change in cardiac function after mitral valve replacement[J]. 解放军医学杂志（英文版）, 1992,(3):256-260. DOI：

[1]钱伟,张宝仁,朱家麟,陈如坤,郝家骅,蔡凯华,刘振华.Mechanism of change in cardiac function after mitral valve replacement[J].Journal of Medical Colleges of PLA,1992(03):256-260. DOI：

摘要

Abstract

<正> Left ventricular contractility was measured using Doppler echocardiography in 37 pa-tients with rheumatic mitral valve disease.Myocardial pathological lesions were studied quanti-tatively by means of the image analysis system and light microscopic examination.The resultsshowed that attenuated cardiac function is due to pathological change of the mitral valve andpathological lesions of the left ventrieular myocardium including increased interstitial fibrosis

thickened intramyocardial arterial wall leading to concentric narrowing of the lumen

hyper-trophic myocardial fiber and endocardial proliferation.Since mitral valve replacement can notcorrect the irreversible myocardial pathological lesions

perioperative protection of the my-ocardium should be emphasized

and intraoperative injury to the myocardium should be mini-mized as far as possible.

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